Journal Article

PUBDB-2025-02590

http://join2-wiki.gsi.de/foswiki/pub/Main/Artwork/join2_logo100x88.png Publisher Correction: Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis

Reichelt, J. ; Sachs, W. ; Frömbling, S. ; Fehlert, J. ; Studencka-Turski, M. ; Betz, A. ; Loreth, D. ; Blume, L. ; Witt, S.Extern*CSSB*DESY* ; Pohl, S. ; Brand, J. ; Czesla, M. ; Knop, J.Extern*EMBL* ; Florea, B. I. ; Zielinski, S. ; Sachs, M. ; Hoxha, E. ; Hermans-Borgmeyer, I. ; Zahner, G. ; Wiech, T. ; Krüger, E. ; Meyer-Schwesinger, C. (Corresponding author)

2023
Springer Nature [London]

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Please use a persistent id in citations: doi:10.1038/s41467-023-38206-0  doi:10.3204/PUBDB-2025-02590

Abstract: The original version of this Article contained errors in Figures 3b, 7d, 7e and 8b, where boxes were used instead of arrows to indicate the specific signals for the blots, and in Figure 8c, the format of the “betas“ was incorrect. This has been corrected in both the PDF and HTML versions of the Article.

Note: cc-by, The authors would like to thank Antonio Virgillio Failla from the UMIF,UKE and Roland Thuenauer from the ALFM, CSSB, DESY for technicalassistance in super resolution microscopy.

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Contributing Institute(s):

1. Strukturelle Mikrobiologie CSSB (FS-CS)
2. CSSB - Core Facility - Protein Production (CSSB-CF-PP)

Research Program(s):

1. 633 - Life Sciences – Building Blocks of Life: Structure and Function (POF4-633) (POF4-633)

Experiment(s):

1. No specific instrument

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Linked articles:

http://join2-wiki.gsi.de/foswiki/pub/Main/Artwork/join2_logo100x88.png Journal Article Reichelt, J. ; Sachs, W. ; Frömbling, S. ; Fehlert, J. ; Studencka-Turski, M. ; Betz, A. ; Loreth, D. ; Blume, L. ; Witt, S.CSSB*DESY* ; Pohl, S. ; Brand, J. ; Czesla, M. ; Knop, J.Extern*EMBL* ; Florea, B. I. ; Zielinski, S. ; Sachs, M. ; Hoxha, E. ; Hermans-Borgmeyer, I. ; Zahner, G. ; Wiech, T. ; Krüger, E. ; Meyer-Schwesinger, C. (Corresponding author)
Non-functional ubiquitin C-terminal hydrolase L1 drives podocyte injury through impairing proteasomes in autoimmune glomerulonephritis
Nature Communications 14(1), 2114 (2023) [10.1038/s41467-023-37836-8]2023     Files BibTeX | EndNote: XML, Text | RIS

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