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| 001 | 89789 | ||
| 005 | 20250731122401.0 | ||
| 020 | _ | _ | |a 9780819476357 |
| 024 | 7 | _ | |a pmid:17645476 |2 pmid |
| 024 | 7 | _ | |a 1996-756X |2 ISSN |
| 024 | 7 | _ | |a 0277-786X |2 ISSN |
| 024 | 7 | _ | |a 10.1117/12.822292 |2 doi |
| 024 | 7 | _ | |a WOS:000299776200021 |2 WOS |
| 024 | 7 | _ | |a openalex:W2021869158 |2 openalex |
| 037 | _ | _ | |a PHPPUBDB-15429 |
| 041 | _ | _ | |a eng |
| 082 | _ | _ | |a 620 |
| 100 | 1 | _ | |a Vielhauer, S. |
| 110 | 1 | _ | |a DESY |b Experiments with synchrotron radiation |
| 111 | 2 | _ | |a SPIE Europe Optics and Optoelectronics |c Prague |d 2009-04-20 - 2009-04-23 |w Czech Republic |
| 245 | _ | _ | |a Phosphor materials under high-density XUV FEL excitation: mechanisms of luminescence quenching |
| 260 | _ | _ | |a Bellingham, Wash. |b SPIE |c 2009 |
| 295 | 1 | 0 | |a Damage to VUV, EUV, and X-Ray Optics II (Proceedings Volume ) |
| 300 | _ | _ | |a 73610R-1 - 73610R-9 |
| 336 | 7 | _ | |0 33 |2 EndNote |a Conference Paper |
| 336 | 7 | _ | |2 BibTeX |a INPROCEEDINGS |
| 336 | 7 | _ | |2 DRIVER |a conferenceObject |
| 336 | 7 | _ | |0 PUB:(DE-HGF)16 |2 PUB:(DE-HGF) |a Journal Article |m journal |
| 336 | 7 | _ | |0 PUB:(DE-HGF)8 |2 PUB:(DE-HGF) |a Contribution to a conference proceedings |b contrib |m contrib |
| 336 | 7 | _ | |0 PUB:(DE-HGF)7 |2 PUB:(DE-HGF) |a Contribution to a book |m contb |
| 440 | _ | 0 | |a SPIE Proceedings |v 7361 |
| 500 | _ | _ | |3 Converted on 2013-05-30 13:46 |
| 500 | _ | _ | |3 Converted on 2013-06-21 19:20 |
| 520 | _ | _ | |a As liver fibrosis is the result of persistent necroinflammation in the liver, pro-inflammatory cytokines secreted in response to cell injury have a central role in the pathogenesis of liver fibrosis. We aimed to investigate the association of cytokine gene polymorphism and liver fibrosis among Chinese patients with chronic hepatitis B.Polymorphisms at interleukin-10 (IL-10-627, -1117), interleukin-1-beta (IL-1beta-511, -31, -3964), interleukin-1 receptor antagonist (IL-1RN), and tumor necrosis factor-alpha (TNF-alpha-308, -238) among Chinese chronic hepatitis B patients were determined. Severe liver fibrosis was defined as Ishak fibrosis score = 4 (of 6).Fifty-nine of 273 (22%) patients had severe fibrosis. The distribution of genotypes for IL-10-627 was CC (11%), CA (41%), and AA (48%). The CC genotype at IL-10-627 was protective against severe fibrosis (odds ratio (OR) 0.11; 95% CI 0.014-0.82; P = 0.032). After adjusted for baseline variables, the adjusted OR of CC genotypes at IL-10-627 for severe fibrosis was 0.063 (95% CI 0.06-0.64; P = 0.063). Other gene polymorphisms at IL-1beta, IL-1RN, TNF-alpha, and IL-10 had no significant association with severe fibrosis. Weak linkage disequilibrium was observed between IL-10-627 and IL-10-1117 with linkage disequilibrium coefficient of 0.12 (P < 0.001). The distribution of haplotypes of IL-10-1117 and IL-10-627 was A-A (69%), A-C (26%), and G-C (5%). High and intermediate IL-10 production (A-C and G-C) haplotypes were protective against severe fibrosis (OR 0.62; 95% CI 0.39-0.99; P = 0.046).High production genotype and haplotypes of IL-10 were associated with less severe liver fibrosis in chronic hepatitis B in Chinese. |
| 536 | _ | _ | |0 G:(DE-H253)POF2-FLASH-20130405 |f POF II |x 0 |c POF2-FLASH-20130405 |a Facility (machine) FLASH (POF2-FLASH-20130405) |
| 536 | _ | _ | |0 G:(DE-H253)II-20022043-EC |a FS-Proposal: II-20022043 EC (II-20022043-EC) |c II-20022043-EC |x 1 |
| 588 | _ | _ | |a Dataset connected to Pubmed |
| 650 | _ | 7 | |0 0 |2 NLM Chemicals |a IL1RN protein, human |
| 650 | _ | 7 | |0 0 |2 NLM Chemicals |a Interleukin 1 Receptor Antagonist Protein |
| 650 | _ | 7 | |0 0 |2 NLM Chemicals |a Interleukin-1beta |
| 650 | _ | 7 | |0 0 |2 NLM Chemicals |a Tumor Necrosis Factor-alpha |
| 650 | _ | 7 | |0 130068-27-8 |2 NLM Chemicals |a Interleukin-10 |
| 650 | _ | 2 | |2 MeSH |a Adult |
| 650 | _ | 2 | |2 MeSH |a Female |
| 650 | _ | 2 | |2 MeSH |a Hepatitis B, Chronic: complications |
| 650 | _ | 2 | |2 MeSH |a Humans |
| 650 | _ | 2 | |2 MeSH |a Interleukin 1 Receptor Antagonist Protein: genetics |
| 650 | _ | 2 | |2 MeSH |a Interleukin-10: genetics |
| 650 | _ | 2 | |2 MeSH |a Interleukin-1beta: genetics |
| 650 | _ | 2 | |2 MeSH |a Liver Cirrhosis: complications |
| 650 | _ | 2 | |2 MeSH |a Liver Cirrhosis: genetics |
| 650 | _ | 2 | |2 MeSH |a Male |
| 650 | _ | 2 | |2 MeSH |a Polymorphism, Genetic |
| 650 | _ | 2 | |2 MeSH |a Tumor Necrosis Factor-alpha: genetics |
| 693 | _ | _ | |0 EXP:(DE-H253)FLASH(machine)-20150101 |1 EXP:(DE-H253)FLASH-20150101 |5 EXP:(DE-H253)FLASH(machine)-20150101 |a FLASH |e Facility (machine) FLASH |x 0 |
| 700 | 1 | _ | |a Babin, V. |
| 700 | 1 | _ | |a de Grazia, M. |
| 700 | 1 | _ | |a Feldbach, E. |
| 700 | 1 | _ | |a Kirm, M. |
| 700 | 1 | _ | |a Nagirnyi, V. |
| 700 | 1 | _ | |a Vasilev, A. |
| 700 | 1 | _ | |a Juha, L. |
| 700 | 1 | _ | |a Bajt, S. |
| 700 | 1 | _ | |a Sobierajski, R. |
| 773 | _ | _ | |0 PERI:(DE-600)2398361-9 |a 10.1117/12.822292 |g Vol. 7361, p. 73610R-1 - 73610R-9 |p 73610R-1 - 73610R-9 |q 7361<73610R-1 - 73610R-9 |t Proceedings of SPIE |v 7361 |x 1996-756X |y 2009 |
| 856 | 4 | _ | |u https://bib-pubdb1.desy.de/record/89789/files/Vielhauer_etal_Phosphors_XUV_FEL.pdf |y OpenAccess |
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| 913 | 1 | _ | |0 G:(DE-HGF)POF2-54G16 |1 G:(DE-HGF)POF2-540 |2 G:(DE-HGF)POF2-500 |9 G:(DE-H253)POF2-FLASH-20130405 |b Struktur der Materie |l Forschung mit Photonen, Neutronen und Ionen (PNI) |v FLASH |x 0 |a DE-H253 |
| 914 | 1 | _ | |y 2010 |
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| 920 | _ | 1 | |i Experiments with synchrotron radiation |k HASYLAB |
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