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@ARTICLE{Kotlarek:632973,
author = {Kotlarek, Daria and Dudek, Katarzyna and Woźniak, Bartosz
and Pastok, Martyna and Shishov, Dmitrii and Cottens,
Sylvain and Biśta, Michał and Krzywiecka, Emilia and
Górecka-Minakowska, Karolina and Jurczak, Kinga and Drmota,
Tomas and Adamczyk, Justyna and Faliński, Szymon and
Gajewska, Daria and Klejnot, Marta and Król, Aleksandra and
Cuprych-Belter, Monika and Mames, Iwona and Mathieu, Arnaud
and Podkówka, Aleksandra and Przytulski, Kamil and Skowron,
Alicja N. and Sypień, Magdalena and Takagi, Toshimitsu and
Wanat, Weronika and Wierzbicki, Igor H. and Wisniewski,
Janusz and Szlachcic, Anna and Pokładek, Ziemowit and
Walczak, Michał J.},
title = {{E}xploration of chemical probes and conformational
flexibility of {GID}4 - the substrate receptor of human
{CTLH} {E}3 ligase complex},
address = {Biorxiv},
reportid = {PUBDB-2025-02299},
year = {2025},
note = {Door proposal: P-20010353; INDU-22-L03 - Captor
Therapeutics Inc., long-term agreementThe project nr
POIR.01.01.01-00-0931/19 “Development of an integrated
technology platform in the field of targeted protein
degradation and its implementation to the pharmaceutical
market” was co-financed by the European Regional
Development Fund.},
abstract = {The application of targeted protein degradation (TPD) is
currently constrained by the limited availability of
low-molecular-weight molecules that can recruit E3 ligases
other than CRBN (Cereblon) or VHL (Von Hippel-Lindau
ligase). In this study, we present the structure-based drug
design (SBDD) of high-affinity ligands that engage E3 ligase
GID4 (Glucose-induced degradation protein 4) in biophysical
and cellular experiments. Through structural studies and
molecular modeling, we identified three groups (clusters) of
compounds that induce distinct conformations of GID4. We
identified potential exit vectors and used the most
promising ligand as a building block to prepare bifunctional
degraders in the form of proteolysis-targeting chimeras
(PROTACs). Although ternary complex formation was successful
in vitro, degradation of BRD4 was not observed, highlighting
the need for further optimization of the degraders. We also
theoretically investigated the likelihood of the identified
GID4 conformations participating in protein-protein
interactions mediated by molecular glue mechanisms. We
believe the expanded ligand diversity discovered in this
study may pave the way for tuning the selectivity and
efficacy of protein-protein interactions involving GID4 and
its neosubstrates.},
cin = {DOOR ; HAS-User},
cid = {I:(DE-H253)HAS-User-20120731},
pnm = {6G3 - PETRA III (DESY) (POF4-6G3)},
pid = {G:(DE-HGF)POF4-6G3},
experiment = {EXP:(DE-H253)P-P11-20150101},
typ = {PUB:(DE-HGF)25},
doi = {10.1101/2025.07.01.662521},
url = {https://bib-pubdb1.desy.de/record/632973},
}