TY - EJOUR
AU - Kwiatkowksi, Anna
AU - Caserta, Giorgio
AU - Schulz, Anne-Christine
AU - Frielingsdorf, Stefan
AU - Pelmenschikov, Vladimir
AU - Weisser, Kilian
AU - Belsom, Adam
AU - Rappsilber, Juri
AU - Sergeev, Ilya
AU - Limberg, Christian
AU - Mroginski, Maria-Andrea
AU - Zebger, Ingo
AU - Lenz, Oliver
TI - ATP-triggered Fe(CN)<sub>2</sub>CO synthon transfer from the maturase HypCD to the active site of apo-[NiFe]-hydrogenase
M1 - PUBDB-2024-05723
PY - 2024
N1 - Funding: Deutsche Forschungsgemeinschaft (DFG, German Research Foundation) through SPP 1927 “Iron Sulfur for Life” project no 311062227; the cluster of excellence ‘UniSysCat’ under Germany’s Excellence Strategy-EXC2008-390540038. EU: the COST Action FeSImmChemNet, CA21115, supported by COST (European Cooperation in Science and Technology).
AB - [NiFe]-hydrogenases catalyze the reversible activation of H<sub>2</sub> using a unique NiFe(CN)<sub>2</sub>CO metal site, which is assembled by a sophisticated multi-protein machinery. The [4Fe–4S]-cluster-containing HypCD complex, which possesses an ATPase activity with an hitherto unknown function, serves as the hub for the assembly of the Fe(CN)<sub>2</sub>CO sub-fragment. HypCD is also thought to be responsible for the subsequent transfer of the iron fragment to the apo-form of the catalytic hydrogenase subunit, but the underlying mechanism remained unexplored. Here, we performed a thorough spectroscopic characterization of different HypCD preparations using infrared, Mössbauer and NRVS spectroscopy, revealing molecular details of the coordination of the Fe(CN)<sub>2</sub>CO fragment. Moreover, biochemical assays in combination with spectroscopy, AlphaFold structure predictions, protein-ligand docking calculations and crosslinking MS deciphered unexpected mechanistic aspects of the ATP requirement of HypCD, which we found to actually trigger the transfer of the Fe(CN)<sub>2</sub>CO fragment to the apo-hydrogenase.
LB - PUB:(DE-HGF)25
DO - DOI:10.26434/chemrxiv-2024-3dx2g
UR - https://bib-pubdb1.desy.de/record/614022
ER -