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@ARTICLE{Listian:613730,
author = {Listian, Stevanus A. and Mazur, Anna-Carina and Kol,
Matthijs and Ufelmann, Edwin and Eising, Sebastian and
Fröhlich, Florian and Walter, Stefan and Holthuis, Joost C.
M. and Barisch, Caroline},
title = {{C}omplex {S}phingolipid {P}rofiling and {I}dentification
of an {I}nositol-{P}hosphorylceramide {S}ynthase in
{D}ictyostelium discoideum},
journal = {iScience},
volume = {27},
number = {9},
issn = {2589-0042},
address = {St. Louis},
publisher = {Elsevier},
reportid = {PUBDB-2024-05618},
pages = {110609},
year = {2024},
abstract = {Dictyostelium discoideum is a professional phagocyte
frequently used to study cellular processes underlying the
recognition, engulfment, and infection course of microbial
pathogens. Sphingolipids are abundant components of the
plasma membrane that bind cholesterol, control membrane
properties, participate in signal transmission, and serve as
adhesion molecules in recognition processes relevant to
immunity and infection. By combining lipidomics with a
bioinformatics-based cloning strategy, we show here that D.
discoideum produces phosphoinositol-containing sphingolipids
with predominantly phytoceramide backbones. Cell-free
expression of candidate inositol-phosphorylceramide (IPC)
synthases from D. discoideum enabled identification of an
enzyme that selectively catalyzes the transfer of
phosphoinositol from phosphatidylinositol onto ceramide. The
IPC synthase, DdIPCS1, shares multiple sequence motifs with
yeast IPC and human sphingomyelin synthases and localizes to
the Golgi apparatus as well as the contractile vacuole of D.
discoideum. These findings open up important opportunities
for exploring a role of sphingolipids in phagocytosis and
infection across major evolutionary boundaries.},
cin = {CSSB-FZB-CB},
ddc = {050},
cid = {I:(DE-H253)CSSB-FZB-CB-20230819},
pnm = {899 - ohne Topic (POF4-899) / SFB 1557 P01 - Umbau und
Nutzung der Lipid-Trafficking-Maschinerie der Wirtszelle
durch pathogene Mykobakterien (P01) (516902060) / SFB 1557
P07 - Untersuchung des Zusammenspiels zwischen der
Plastizität vakuolärer Kontaktstellen und der
Sphingolipid-Homöostase (P07) (516904847) / DFG project
G:(GEPRIS)531703706 - Entschlüsselung der molekularen
Mechanismen des Austritts von Orientia tsutsugamushi aus der
Wirtszelle (531703706)},
pid = {G:(DE-HGF)POF4-899 / G:(GEPRIS)516902060 /
G:(GEPRIS)516904847 / G:(GEPRIS)531703706},
experiment = {EXP:(DE-H253)ALFM-20250101},
typ = {PUB:(DE-HGF)16},
pubmed = {pmid:39286488},
UT = {WOS:001307900600001},
doi = {10.1016/j.isci.2024.110609},
url = {https://bib-pubdb1.desy.de/record/613730},
}