% IMPORTANT: The following is UTF-8 encoded. This means that in the presence
% of non-ASCII characters, it will not work with BibTeX 0.99 or older.
% Instead, you should use an up-to-date BibTeX implementation like “bibtex8” or
% “biber”.
@ARTICLE{Pazicky:472140,
author = {Pazicky, Samuel and Alder, Arne and Mertens, Haydyn and
Svergun, Dmitri and Gilberger, Tim and Loew, Christian},
title = {{N}-terminal phosphorylation regulates the activity of
{G}lycogen {S}ynthase {K}inase 3 from {P}lasmodium
falciparum},
reportid = {PUBDB-2021-04863},
year = {2021},
abstract = {As the decline of malaria cases stalled over the last five
years, novel targets in Plasmodium falciparum are necessary
for the development of new drugs. Glycogen Synthase Kinase
(PfGSK3) has been identified as a potential target, since
its selective inhibitors were shown to disrupt the
parasite’s life cycle. Here, we show that PfGSK3 exhibits
autophosphorylation, leading to an extensive phosphorylation
both in vitro and in the parasite. In the uncanonical
N-terminal region of the parasite enzyme, we identified
several autophosphorylation sites that regulate the activity
of PfGSK3. By combining molecular modeling with experimental
small-angle X-ray scattering data, we show that increased
PfGSK3 activity is promoted by conformational changes in the
PfGSK3 N-terminus, triggered by N-terminal phosphorylation.
Our work provides novel insights into the structure and
regulation of the malarial PfGSK3.},
cin = {EMBL / EMBL-User / CSSB-EMBL / CSSB-EMBL-CL /
CSSB-BNITM-TG},
cid = {I:(DE-H253)EMBL-20120731 / I:(DE-H253)EMBL-User-20120814 /
I:(DE-H253)CSSB-EMBL-20141216 /
I:(DE-H253)CSSB-EMBL-CL-20210806 /
I:(DE-H253)CSSB-BNITM-TG-20210520},
pnm = {6G3 - PETRA III (DESY) (POF4-6G3)},
pid = {G:(DE-HGF)POF4-6G3},
experiment = {EXP:(DE-H253)P-P12-20150101},
typ = {PUB:(DE-HGF)25},
doi = {10.1101/2021.06.18.448949},
url = {https://bib-pubdb1.desy.de/record/472140},
}